In addition, delayed onset of the tibialis anterior (TA) has also been observed in people with CAI, as well as an impaired balance in TA-PL co-activation ratio in response to sudden inversion perturbations or pre-landing phases of running and stop-jump maneuvers. Recently, significant differences in preparatory activity displayed by peroneus longus (PL) have been reported in pre-landing scenarios in basketball and volleyball players with CAI, with decreased PL EMG amplitudes just before the foot contact to the ground. Specifically, these studies have shown lower pre-activation and delayed onset of the peroneal muscles during dynamic tasks in people with CAI, which they identified as contributing factors in the etiology of future ankle sprains. Studies utilising electromyography (EMG) have revealed changes in the activation of the stabilizer muscles of the ankle following injury. Īs a result, a wide variety of altered feedback/feed-forward mechanisms has been shown in subjects with CAI, but it remains uncertain whether these deficits are due to local or spinal/supraspinal conditions. CAI is associated with deficits in postural, neuromuscular, and sensorimotor control and is often bilateral. CAI is associated with repetitive lateral ankle instability episodes, where both functional and mechanical insufficiencies predispose to multiple sprains. Given the intrinsic characteristics of basketball practice and its specific physical demands, more than 70% of basketball players who suffer an acute ankle sprain will develop recurrent sprains between 6 weeks and 18 months after the initial injury, with approximately 20–40% developing chronic ankle instability (CAI). These results showed improvements in feedback/feed-forward strategies following DN, including enhanced neuromuscular control and static postural control, with the potential to become a convenient and accessible preventive treatment in CAI subjects.Īfter an initial ankle sprain, anatomical changes such as laxity, impaired arthrokinematics, or synovial changes can lead to joint insufficiencies that predispose to recurrent ankle sprains. Significant reductions in the ML and AP displacements and sway variability of CoP were found for the DN group. The DN group displayed a significant increase in PL and TA pre-activation values, which were maintained 1 month later. Measures were obtained prior to a single DN intervention, immediately after, at 48 h, and 1 month after. Center of pressure (CoP) displacement and sway variability in anterior-posterior (AP) and medio-lateral (ML) directions were measured with a force platform during a single leg balance test (SLBT). Pre-activation amplitudes of PL and TA were assessed with surface electromyography (EMG) during a dynamic landing test. Thirty-two male and female basketball players with CAI were randomly assigned to receive either DN ( n = 16) or placebo DN ( n = 16). A single-blinded randomized controlled trial was conducted. This study aimed to compare the effects of dry needling (DN) versus placebo DN applied to the peroneus longus (PL) and tibialis anterior (TA) on neuromuscular control and static postural control in basketball players with chronic ankle instability (CAI).
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